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Hepatitis C virus negative patients receiving more than the blood of hepatitis C virus infection after the vesting


Are exactly the same as in the blood B 43,[link widoczny dla zalogowanych],84,96 and 147 days after blood can be detected in non-dominant gene IaHCV, and blood donor A, Hcv homologous (97% to 100% homology). Case 2 blood recipients who received three blood donors were infected with gene 1a (A: ALT as 64W/val, I {cvRNA to 2.3 × lOSge/rn1),[link widoczny dla zalogowanych], 2Jo 【B: ALT-shaped 35 / rnl, HCVRNA to 34xlge/rn1) and 1b (c: A for the 5OlU/ml,[link widoczny dla zalogowanych], HCVRNA to 2.2 × lO5ge/m1). 14d recipients in blood transfusion HCVRNA positive, dominant gene to 1a, and donors A genetically identical (995% homology). In the first 61 days of post-transfusion non-optimal detection of gene 1b, and homologous blood donors cHCV. 3 patients received the first three recipients of blood donors, three donors were infected with HCV genotype 1b (A: ALT was 201U/ml, HCVRNA for the 30 × lOSge / rrd; B: ALT as 4IU/rnl, HCVRNA for the 83 × 1oSge/rnl; C: ALT as 38IU/ml,[link widoczny dla zalogowanych], HCVRNA to 23.0 × to5ge/rn1). 20d recipients in blood transfusion HCVPLNA positive, genotype lb, with donor A gene homologous with the B, c different source (95.5%, 96% homology). 4 down 2 recipients received blood donors,[link widoczny dla zalogowanych], HCV genotypes were 1a (A: ALT as 11ILl/nil, HCVRNA to 4.7 × lO5ge / m]) and 1b 【B: ALT as 11ILl/rnl, HCVRNA was <2.0 × 10'ge / m1). 8d recipients in blood transfusion HCVRNA positive, genotype la, A gene homologous with the donor (99% homology). To transfusion 94d, recipient genotypes did not change, ALT normal gradually, most blood samples HCVRNA negative. 2 blood samples after HCVRNA positive (126d and 5d), genotype 1b, and homologous blood donor B (100% homology). The last 2 specimens (265d and 283d) HCVRNA were negative. The results show that. HCV infection can result in hybrid with a genotype was the dominant gene of mixed infection of multiple genes. HCV of different genes compete with each other to ask each other inhibited the formation of a single gene dominant gene, the subject of mixed infection of multiple genes. The authors believe that HCV infection in mixed dominant gene of choice, is not immune factors play an important role in the virus itself is the key, and the level of blood donors and blood transfusion viral replication interval may be related. Whether congenital factors is not clear. (Abstract CHEN Shi-Zhu Zhang Jin Hong Yang Kanghu school) 013HLAA2/6802 parent gene and the human immunodeficiency virus type 1 perinatal decreased risk of infection related to (English] / Mae_Do ~ clKS ... / / IrffectDis .. 2001183 (3) .. 503 - 5O6 HLAA2/6802 gene on the mother for the study of a child transmission of human immunodeficiency virus type 1 (HIV-I) the impact of choice of the University of Nairobi HIV-1 perinatal transmission research center Hrv-|-infected mothers and their children as research object, all objects were detected in HLA-1 gene. a total of 135 of 171 mothers were studied harp, the results of which l9 person is a perinatal infection, 152 uninfected human at birth, but 21 were later breastfeeding in the infected HIV.1. HLAA2/6802 Kisii {; [i of 72 children, 2 (28%) occurred in the perinatal period HIV.1 infection. HLAA2/6802 gene was significantly lower than negative 99 occurred in children l7 cases (17.2) HIV-1 infection (OR = 0.17, P = 0005). HLAA2/6802 the presence of genes does not protect breastfeeding from I-IP / A 1 infection, HLAA2/6802 gene positive people are 1O to 72 people (i3.9%) infected through breastfeeding HW.1, while gene-negative HLAA2/6802 11 people 99 people (111%) were infected with HIV .1 (0 = 1.1, P = 0.99). multi-factor l ~ gist / c regression
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